By John Elefteriades
Protecting the pathophysiology, imaging, analysis, and therapy of numerous aortic aneurysms and dissections, this resource is helping physicians successfully research and overview affected participants in medical or emergency care settings. delivering a big selection of illustrations, x-rays, and operative photos to stress key anatomic observations, this advisor comprises state of the art perception at the newest biologic, radiologic, medical, and surgical advancements that experience taken position within the box. awarded in a reader-friendly layout, this resource offers end-of-chapter questions and a point-counterpoint layout to investigate differing views from popular specialists on those ailments. The Q
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Atherosclerosis is the main cause of 435aortic aneurysms (23,24). The intima of the aorta shows massive fibrosis and calcification, and increased amounts of extracellular fatty acids. The integrity of this layer can be compromised by the extracellular matrix degraded by histiocytic cells, while additional degenerative changes can develop within the fibrous tissue. These mechanisms may lead to intimal rupture, most often at the edges of plaques. Intimal thickening increases the distance between the endothelial layer and the media, compromising the nutrient and oxygen supply.